Glomerulo nefritis acute is the term widely used, which refers to a group of kidney diseases in which inflammation occurs in glamerulus. (Brunner and Suddarth, 2001).
Glamerulo nefritis is peradanga and damage to the equipment filters the blood capillaries while kidney (Glamerulus), (Japaries, willie, 1993).
Glamerulus nefritis syndrome is a ditadai by inflammation of glemerulus followed by the establishment of several antigens (Engran, Barbara, 1999).
II.Etiologi
Streptococcus bacteria.
Communication with auto-immune diseases other.
Reaction drugs.
Bacteria.
Virus.
III.Manifestasi Clinic
Faringitis or tansiktis.
Fever
Headache
Malaise.
Pelvic pain
Hypertension
Anoreksi
Vomiting
Ederma acute
Oliguri
Proteinuri
Brown-colored urine.
IV.Patofisiologi
Prokferusi cellular (peningkata cell production endotel; is yag stucco glomerulus), infilaltrasi lekosit to glameruus, and penebalan membranes or membrane filbtrasi glamerulus basalt generate network kehilagan surface of the stomach and filters. In glamerulo nefritis acute kidney swell, swelling and congestion.
In fact the case, the reaction is the stimulation of infection by bacteria in the steeptococus A tengorok, which biasayang precede glomerulo nefritis intervals until 2 - 3 weeks. Product streptacocus act as antinge, bersirkulasi stimulating antibody that causes kidney injuries.
Dealing medical V.
Anti hipertensif
Anti dkurektik
Infection with antibiotics streptokokal set.
Anti-Biotic profilaktif during pemuliha
Enter and output.
TTV 2 - 4 hours.
Sodium restriction.
VI.Pemeriksaan diagnostic
Urinalisis (UA) shows gross hematnya, protein and dismonfik (not suited) human resources, leusit, and gypsum hialin.
Rows filtration glomeruslus (IFG) meurun, klerins kreatinin on unrin used as a gauge spesine urine and LFG gathered 24 hours. Samples of blood for kreatinin also be featured with the way games all around flow (midstream).
Blood urea nitrogen (BUN) and serum menigkat when kreatinin kidney function started to decline.
Albumin and serum total protein may be normal or slightly down (because hemodilusi).
Example eletrokoresisi to random urine protein mengidenti filaasi types of urine protein in the urine issued.
What electrolyte Sodium and shows improvement or increase the normal rates, and potassium chloride.
VII.Potensial complications
Hypertension.
Dekopensasi heart
GGA (Kidney Failure Akut)
Nursing VIII.Asuhan
Assessments.
1.Identitas patients.
2.Riwayat disease, first, and now the family.
3.Riwayat / of the risk factors.
a.Bagaimana frequency miksinya, whether there are:
b.Adakah aberration time miksi such as:
c.Apakah pain there is in the local area or in Unum.
d.Apakah disease arising after the peyakit the other.
e.Apakah there is nausea and vomiting.
f.Apakah there is oedema.
g.Bagaimana circumstances urinnya (volume, color, smell, weight, type, number of urie in 24 hours).
h.Adakah sekret or blood out.
i.Adakah hambantan sexual.
j.Bagaimana history, menstruation (menache, duration, many, sirkulasinya, keluhannya).
k.Bagaimana Historical pregnancy, arbortus, the use of kontrsepsi.
l.Rasa pain (location, identity, the emergence of pain).
m.Riwayat Maternity.
Bleeding n.Riwayat.
4. Physical data:
Inspection:
in general and specifically in the genital area palpasi:
daeraha on bdomen, jar, fold the thigh.
Auskultasi: blood abdomen.
Percussion: regional abdomen, kidney.
Keadaa general patients:
The level of awareness.
Eliputi vital high tension, pulse, temperature, breathing.
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